Pages that link to "Item:Q166888"

The following pages link to Prion Diseases: Causes and Molecular Basis (Q166888):

Displayed 50 items.

• A dynamic spatio-temporal model for spatial data (Q240131) ‎ (← links)
• Aaron M. Foley (Q241394) ‎ (← links)
• Dennis M. Heisey (Q241897) ‎ (← links)
• Kenneth H. Johnson (Q243142) ‎ (← links)
• Jay R. Schneider (Q243181) ‎ (← links)
• Low copper and high manganese levels in prion protein plaques (Q243200) ‎ (← links)
• In vitro prion protein conversion suggests risk of bighorn sheep (Ovis canadensis) to transmissible spongiform encephalopathies (Q243497) ‎ (← links)
• Michael R. Ebinger (Q243637) ‎ (← links)
• Michael D. Samuel (Q243761) ‎ (← links)
• Paul Slota (Q243948) ‎ (← links)
• Is exposure to cyanobacteria an environmental risk factor for amyotrophic lateral sclerosis and other neurodegenerative diseases? (Q244135) ‎ (← links)
• Red-backed vole brain promotes highly efficient in vitro amplification of abnormal prion protein from macaque and human brains infected with variant Creutzfeldt-Jakob disease agent. (Q244686) ‎ (← links)
• Association mapping of genetic risk factors for chronic wasting disease in wild deer (Q244782) ‎ (← links)
• Using landscape epidemiological models to understand the distribution of chronic wasting disease in the Midwestern USA (Q244868) ‎ (← links)
• Emerging prion disease drives host selection in a wildlife population (Q245952) ‎ (← links)
• Highly efficient amplification of chronic wasting disease agent by protein misfolding cyclical amplification with beads (PMCAb) (Q245974) ‎ (← links)
• The role of genetics in chronic wasting disease of North American cervids (Q246044) ‎ (← links)
• Mapping brucellosis increases relative to elk density using hierarchical Bayesian models (Q247863) ‎ (← links)
• Linking process to pattern: estimating spatiotemporal dynamics of a wildlife epidemic from cross‐sectional data (Q248125) ‎ (← links)
• Bison PRNP genotyping and potential association with Brucella spp. seroprevalence (Q249293) ‎ (← links)
• Modeling routes of chronic wasting disease transmission: Environmental prion persistence promotes deer population decline and extinction (Q250960) ‎ (← links)
• Diversity and distribution of white-tailed deer mtDNA lineages in chronic wasting disease (CWD) outbreak areas in southern Wisconsin, USA (Q251491) ‎ (← links)
• Meat and bone meal and mineral feed additives may increase the risk of oral prion disease transmission (Q251544) ‎ (← links)
• Soil clay content underlies prion infection odds (Q251661) ‎ (← links)
• Homogenization of large-scale movement models in ecology (Q251672) ‎ (← links)
• Persistence of pathogenic prion protein during simulated wastewater treatment processes (Q252047) ‎ (← links)
• Landscape genetics and the spatial distribution of chronic wasting disease (Q252391) ‎ (← links)
• Pathogenic prion protein is degraded by a manganese oxide mineral found in soils (Q259525) ‎ (← links)
• Cognitive and behavioral coping in response to wildlife disease: The case of hunters and chronic wasting disease (Q266907) ‎ (← links)
• Applying a Bayesian weighted surveillance approach to detect chronic wasting disease in white‐tailed deer (Q268013) ‎ (← links)
• Prion seeding activity in plant tissues detected by RT-QuIC (Q269364) ‎ (← links)
• Plants as vectors for environmental prion transmission (Q269481) ‎ (← links)
• Chronic wasting disease: State of the science (Q276403) ‎ (← links)
• Susceptibility of beavers to chronic wasting disease (Q277354) ‎ (← links)
• Deer carcass decomposition and potential scavenger exposure to chronic wasting disease (Q285687) ‎ (← links)
• Landscape features fail to explain spatial genetic structure in white-tailed deer across Ohio, USA (Q285788) ‎ (← links)
• The ecology of chronic wasting disease in wildlife (Q286176) ‎ (← links)
• On the origin of brucellosis in bison of Yellowstone National Park: a review (Q286241) ‎ (← links)
• Institutional trust, beliefs, and evaluation of regulations, and management of chronic wasting disease (CWD) (Q288859) ‎ (← links)
• Genetic susceptibility to chronic wasting disease in free-ranging white-tailed deer: complement component C1q and Prnp polymorphisms (Q289997) ‎ (← links)
• Spatial heterogeneity of prion gene polymorphisms in an area recently infected by chronic wasting disease (Q293357) ‎ (← links)
• Surveillance for transmissible spongiform encephalopathy in scavengers of white-tailed deer carcasses in the chronic wasting disease area of wisconsin (Q294088) ‎ (← links)
• Increased attack rates and decreased incubation periods in raccoons with chronic wasting disease passaged through meadow voles (Q296008) ‎ (← links)
• Chronic wasting disease alters the movement behavior and habitat use of mule deer during clinical stages of infection (Q297358) ‎ (← links)
• Epidemiological differences between sexes affect management efficacy in simulated chronic wasting disease systems (Q300816) ‎ (← links)
• Examination of the interaction between age-specific predation and chronic disease in the Greater Yellowstone Ecosystem (Q301715) ‎ (← links)
• Spatial and temporal patterns of chronic wasting disease: Fine-scale mapping of a wildlife epidemic in Wisconsin (Q301793) ‎ (← links)
• Ultraviolet-ozone treatment reduces levels of disease-associated prion protein and prion infectivity (Q307694) ‎ (← links)
• Department for Environment Food and Rural Affairs (Q331695) ‎ (← links)
• National Wildlife Health Center (Q331853) ‎ (← links)